Coronary Artery Disease - Pathophysiology and Management Research Paper

Coronary Artery unsoundness - Pathophysiology and Management - Research Paper Example plague is a component of fat, cholesterol and separate substances of the blood. Plague is slowly built up over several years forming a condition known as atherosclerosis (Cohen & Hasselbring, 2007). After a long period of building up, plague hardens and narrows coronary artery booster cable to limits of flow of oxygenated blood to the heart. As the plague hardens, it eventually ruptures, causing a blood clot, a role that can lead to blockage of blood flow through the coronary artery. If the flow of oxygenated blood is lessen or blocked, a heart attack occurs (Cohen & Hasselbring, 2007). The beginning of manifestationatic hotdog is normally about 10 years in men, but CAD cases in women are more prevalent especially to women who contrive reached menopause. However, it is believed that CAD cases tend to cumulate in families (Cohen & Hasselbring, 2007). Studies have indicated that history of CA D risks in families with CAD cases is common. Although some(a) people can argue that CAD is a hereditary disease, some factors like other health complications such as hypertension, high blood pressure and obesity facilitates risks of CAD. All these are widely associated with aliment styles. There are precautions that physicians give to patients suffering from CAD. Unfortunately, many people suffering from CAD fail to strike these precautions seriously (Cohen & Hasselbring, 2007). For example, the habit of smoking has become so rampant with over twenty five percentage of people with over eighteen years smoking cigarette. Smoking is said to multiply the effects of other CAD factors. It is estimated to cause twenty percent of CAD deaths. The theory of aetherorosclerosis states that the process symbolizes an effort to heal in response to endothelial injury (Cohen & Hasselbring, 2007). During this process, the first step involves development of fatty streaks. These streaks tend to fo rm among endothelium and internal elastic lamina. In the recent past, inflammation is becoming a component of atherosclerosis genesis and plague instability. Patients with CAD are at danger of risk factors known as metabolic syndrome (Cohen & Hasselbring, 2007). CAD is known for its persistent burden to many people all over the world. However, its risks can be managed if skill of lipid- humbleing models is followed. The first lipid- lowering therapy focuses on therapeutic lifestyle changes (Cohen & Hasselbring, 2007). This includes dietary changes, constant physical activities, avoiding precarious practices like smoking and alcohol drinking, and undertaking recommended wait lose. However, there are pharmacologic agents that help right lipid levels although, therapeutic lifestyle is preferred to them (Cohen & Hasselbring, 2007). Patients with CAD are carefully examined. The examination includes description of the pain, its spot and severity (Cohen & Hasselbring, 2007). Diagnos is of CAD involves detailed patients history and electrocardiogram. After the initial diagnosis, laboratory tests follow. Angina is the commonest symptom although some patients remain asymptomatic. Some of the notable symptoms include chest pain, sweating, and yellowish tumors at either lower or upper lids. One of the major causes of CAD is stress (Cohen & Hasselbring, 2007). In this regard, nursing care mean involves helping the patient to realize the cause of the disease and how best they can refrain from it. Since stress is preponderance to most CAD patients, one of